So what’s in a name you may ask? A lot sometimes, and othertimes, well not so much.

     [Lyme Disease] was originally known as a “relapsing fever” or borreliosis, due to the taxonomy,

(the branch of science concerned with classification, especially of organisms; systematics.

  • the classification of something, especially organisms.),

and nature of the organism that causes the disease. Spirochetes are their own phylum,



  1. a principal taxonomic category that ranks above class and below kingdom.),

 and they shed their outer surface in a mechanism called blebbing.


Through “antigenic variation,” or the ability to modify their outer surface proteins (Osps), the organism causes “relapsing fever” because the host immune response, (your immune system), must constantly address the variable antigens, (an antigen is a harmful substance that causes the body to produce antibodies). For this reason, it is not possible to vaccinate against borrelia.

“1986, Alan Barbour. Spirochetes bleb or pinch off bits to release its surface proteins stealth bombing or turning off the immune system. “It’s using some sort of stealth-bomber-type mechanism,” he says. Or, using another diversionary tactic called blebbing, the spirochete can pinch off bits of its membrane in order to release its surface proteins. “It’s like a bacterial Star Wars defense program.”


     Both the vaccine and Borrelia Burgdorferi contain OspA, a highly toxic immune-suppressing triacyl lipoprotein, (“a lipoprotein is a biochemical assembly whose purpose is to transport hydrophobic lipid (a.k.a. fat) molecules in water, as in blood or extracellular fluid.”).Lipoproteins in bacteria: structures and pathways

 OspA is Pam3cys or a TLR2/1 agonist.

OspA structure: it is a triacyl lipoprotein ,as you can see below, it has 3 acyl groups.


     You got the vaccine.  Everyone with Lyme got LYMErix. This is what LYMErix is, and how this vaccine-was-the-disease works:

It’s Pam3Cys or a triacylated lipoprotein; the degree of acylation is equated with its toxicity. 

So what is acylation?  It’s the zig-zaggy lines that mean Carbon-Carbon-Carbon, yes, hydrocarbons, like margarine or octane. Exactly, the name just refers to the number of carbons in each carboxyl or acyl group.  Palmitic (the Pam in Pam3Cys) has X number of carbons, gasoline, 8, linoleic acids, like 14. Look up what are alkanes then add a COOH group and you have one of these fatty acids.
Something highly acylated like this (3 or more fatty acids hanging off) are managed by Toll-like Receptor (TLR) 2 and TLR1, together. Therefore a “TLR2/1-agonist” is another term that generally refers to lipoproteins like those from Borrelia, mycoplasma, mycobacteria, and others, like Brucella. (But they can manage other compounds.)

Pam3Cys and fungal lipid molecules like it, is shed with these blebs.

“The magnitude of modulation was directly dependent on the quantity of OspA. OspA interferes with the response of lymphocytes to proliferative stimuli including a blocking of cell cycle phase progression”

“It’s the perfect stealth bacteria,” says one frustrated physician. He’s talking about Borrelia burgdorferi, the bacterium that causes Lyme disease. This illness, which is often mistaken for diseases ranging from multiple sclerosis to Lupus, can inflict excruciating headaches and muscle pain, affect the brain and nervous system, attack major organs, and inflame joints…”  —Energy Science News,  [full article: BrookhavenStealthBacteria]

     TLR2/1 agonists cause tolerance, (Immune tolerance. From Wikipedia, the free encyclopedia. … Immune tolerance, or immunological tolerance, or immunotolerance, is a state of unresponsiveness of the immune system to substances or tissue that have the capacity to elicit an immune response in a given organism), and cross tolerance, (extension of the tolerance for a substance to others of the same class, even those to which the body has not been exposed previously), to other TLR’s to no longer detect or fight this or other pathogens. It’s like non-HIV AIDS, where it causes immunosuppression and all the other infections that the immune system was able to keep under control are now able to rear their ugly heads and wreak havoc in your body. From HHV-6, Epstein-barr, Cytomegalovirus, Zoster, Candida, Coxsackievirus, Mycoplasma, Babesiosis, PANDAS (Pediatric Autoimmune Neuropsychiatric Disorders Associated with Streptococcal Infections), etc.. OspA is the culpret in both vaccine and Lyme. It causes permanent immunosuppression. The lymph nodes and brain are targeted, causes chronic encephalopathy, mutated B-cells and “The Great Neuro-Degenerative Imitator”…. ALS, MS, lupus, parkinsons, Autism, ADHD/ADD, dementia, alzhiemers, stroke, cancer, schizophrenia, seizure disorder, Huntington’s, mitochondrial disease, myasthenia gravis, autoimmune encephalitis, chronic fatigue syndrome, fibromyalgia, etc..

The perfect stealth pathogen because immunosuppressed victims produce no to little antibodies (think serongative test results),. The Dearborn fraud changed the testing to a high antibody concentration. So the immune-suppressed neurological B-cell AIDS victims cannot get a positive test nor a diagnosis. This was the whole purpose of Dearborn, it hid their vaccine injuries. The current definition says you can only have Lyme if you have high antibodies and arthritic Lyme with no other symptoms.

CDC 2017 Case Definition

You can’t have a vaccine against a disease known to shed surface antigens that are fungal and known to cause immunosuppression.  Notice there are no vaccines against Tuberculosis or other fungal diseases.  To get around this scientific dilemma, CDC staff who own patents for vector borne diseases (,(American Lyme Disease Foundation, a Non-non profit), simply declared that only the hypersensitivity or HLA-linked allergy outcome would be allowed to be a “case” of “Lyme disease” at a fake consensus conference in Dearborn, Michigan, in 1994. Only 15% of cases test positive.

AND SERONEGATIVE DISEASE (Note that if Wormser owns stock in Abbott- they won’t produce a real
test for Lyme; an Abbott representative was at the Congressional Hearing 120717.):




Here we see Gary Wormser talking about immunosuppression from Lyme and LYMErix, yet how could that be if he also insists the Dearborn standard is real,… and subsequently Klempner’s Non-Retreatment non-study, upon which the

IDSA , (Infectious Disease Society of America),”guidelines” are based? IDSA guidelines for Lyme Disease

The crux of the crime is the  Dearborn diagnostic standard.


Note that Gary Wormser above discusses the mechanism of seronegative Lyme and the resultant immunosuppression
from exposure to OspA either thru blebbing or vaccination, resulting in LYMErix Disease being the same disease as Chronic Lyme/Great Imitators.

Tolerance and Cross Tolerance 
“we propose that TLR2 signaling induces rapid depletion of IRAK1, which impairs IFN-I induction by TLR7/9. This novel mechanism, whereby TLR2 inhibits IFN-I induction by TLR7/9, may shape immune responses to microbes that express ligands for both TLR2 and TLR7/TLR9, or responses to bacteria/virus coinfection.”
B-cells are mutated 
“immature B cells can also be seen in the spinal fluid.  These cells can appear quite atypical- not unlike those of transformed or neoplastic lymphocytes.”

Lymph nodes are targeted
Together, these findings suggest a novel evasion strategy for B. burgdorferi: subversion of the quality of a strongly induced, potentially protective borrelia-specific antibody response via B. burdorferi’s accumulation in lymph nodes.”

Chronic Encephalopathy  
Bacterial lipoproteins can disseminate from the periphery to inflame the brain. “We conclude that some bacterial lipoproteins can disseminate from the periphery to inflame the brain”  ttps://


“Steere’s original, 1986, Lyme-diagnostic serology report which resulted in the 1990 CDC standard (and says Lyme is Relapsing Fever, and that the only valid antibody to use to detect all cases of Lyme was flagellin, or band 41):

A snippet from that 1986 report:

The above was mentioned at the 2001, Jan, FDA Meeting on LYMErix Adverse Events by Kathleen Dickson:  “The standard changed at Dearborn, Lyme used to be considered Relapsing Fever according to Allen Steere, above, people with LYMErix Disease are like chronic Lyme victims, and that could be because of the immunosuppression as per Mario Philipp (OspA results in the production of the immunosuppressive cytokine IL-10) and Ray Dattwyler’s NK cell suppression studies):

1990 CDC Serology standard (same as Steere’s 1986 report; means Lyme is Relapsing Fever):
1990 Case Definition for “Lyme Disease” was “Relapsing Fever”








Dearborn Invitations (connotes consensus, but it was not):, Who Approved:

Dearborn booklet (111 pages) showing no one agreed with Steere’s proposal and who was on all the committees

Gary Wormser’s peer-reviewed assessment of Steere’s Dearborn proposal where he says only 9/59 patients met the Steere/Dearborn  criteria for IgG (85% of cases are missed):

 Page 74 of the Dearborn booklet – Gary Wormser saying the Steere proposal sucked.  Here is the full text of that assessment:

 (missed 85% of the cases). This was deliberate so that they could say that the “vaccine” worked.

So, hopefully by now you can begin to see the BIG picture about what is, and is NOT [Lyme], and why Chronic Lyme is denied.

It doesn’t exist. No disease. No Treatment. NO CURE!

This is why the Lyme Cryme has to be prosecuted.



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RN for 24 years. Activist for TRUTH in Healthcare. Lyme Warrior. Battling this disease since the late 1980's

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